FASLG and neoplasm: The broad range of cytokines (e.g., IFN-γ, perforin, granzyme B) [127,130,145], and activating (e.g., NKp30, FasL) and inhibitory receptors (e.g., killer inhibitory receptor, KIR) located on the cell surface [145,146] allow them to interact with other immune cells or biomolecules which allow them to recognize tumor cells and then induce antitumor effects [145,147].