In addition, TGFβ1‐stimulated MSC were significantly more adherent to liver sections prepared from explanted diseased human livers of hepatitic nature, which was a pool of non‐alcoholic steatohepatitis/alcohol‐related liver disease cases (unstimulated 2.43 ± 0.13 cells/fov vs. stimulated 3.87 ± 0.23; p < 0.000) compared with cholestatic (primary biliary cholangitis/primary sclerosing cholangitis) sections (unstimulated 1.13 ± 0.11 vs. stimulated: 1.77 ± 0.13) and normal tissue (unstimulated 1.43 ± 0.15 vs. stimulated: 1.47 ± 0.16). This evidence concerns the gene TGFB1 and biliary liver cirrhosis.