To investigate the mechanism of TGF-β1 upregulation of NEU3 translation and test if blocking this effect could attenuate pulmonary fibrosis, in this report, we identified proteins in addition to NEU3 in human lung fibroblasts whose levels are increased by TGF-β1 at the level of translation but not transcription, then identified a common motif in the encoding mRNAs. The gene discussed is TGFB1; the disease is pulmonary fibrosis.