In a mouse model of pulmonary fibrosis, RK-33 reduced bleomycin-induced lung inflammation and fibrosis, and lung tissue levels of DDX3, TGF-β1, and NEU3, suggesting that the TGF-β1 upregulation of the translation pathway may be a viable target to inhibit fibrosis. The gene discussed is TGFB1; the disease is pulmonary fibrosis.