The termination of the inflammatory process was supported by a marked decrease in IL-6, TNF-α, and NF-κB, which are known to enhance inflammation in early ALI (Suratt and Parsons 2006; Devaney et al. 2013) and increase anti-inflammatory cytokines IL-10, which was found to reduce neutrophilic activity and inflammatory mediators in ALI in LPS mice model (Inoue 2000) compared to PC group. This evidence concerns the gene IL10 and acute respiratory distress syndrome.