As a result, neutrophils were recruited more efficiently to tumour colonies, showed improved TrisoMab-mediated ADCC and trogocytosis of cancer cells compared to the parental EGFR-specific IgG, However, the improvement over EGFR-specific IgA, whose Fab targets EGFR and own Fcα targets FcαRI/CD89, was limited. The gene discussed is FCAR; the disease is neoplasm.