GALNT3 and lung carcinoma: In the study of Mi So Park et al., it was found that the main mechanism by which the polypeptide N-acetyl-galactosaminyltransferase (GALNT3) inhibited the development and progression of lung cancer in xenograft and syngeneic mouse models was the ability of MDSCs to infiltrate the tumor site and subsequent angiogenesis, thereby inhibiting the development of lung cancer (58).