MIR497HG expression was positively correlated with miR‐195/497 expression and MIR497HG depletion significantly downregulated miR‐195/497 expression in breast cancer cells, supporting previous research that MIR497HG is a precursor of miR‐195 and miR‐497.[13] Moreover, miR‐195/497 abrogated the MIR497HG depletion‐induced promotion of cell proliferation, invasion, migration, and EMT‐like phenotype. The gene discussed is MIR497HG; the disease is breast cancer.