Compared to our T-MN patients, Korean AML patients harbored more somatic mutations in DNMT3A, TET2, NPM1, FLT3, IDH2, KIT, NRAS, CEBPA, and GATA2, but fewer somatic mutations in TP53 and RUNX1 [65, 67]. This evidence concerns the gene FLT3 and acute myeloid leukemia.