Compared to our T-MN patients, Korean AML patients harbored more somatic mutations in DNMT3A, TET2, NPM1, FLT3, IDH2, KIT, NRAS, CEBPA, and GATA2, but fewer somatic mutations in TP53 and RUNX1 [65, 67]. The gene discussed is NPM1; the disease is acute myeloid leukemia.