The general characteristics of HT include diffuse lymphomononocytic cell infiltration; thyroid gland hypertrophy; abnormal thyroid functions; morphological changes in thyroid sonography; increased titers of thyroid autoantibodies, anti-thyroid peroxidase (Anti-TPO), and anti-thyroglobulin (Anti-TG); decreased number of thyroid follicles; and Hurthle cells with granular-pink cytoplasm [3]. The gene discussed is TPO; the disease is hematocrit.