PMVK and hepatocellular carcinoma: Previous studies have shown that accumulated cholesterol stabilizes β‐catenin signaling.[31] But in our study we showed that supplementation of additional cholesterol levels in HCC cells with knockdown PMVK only partially restored cell proliferation levels, suggesting that although cholesterol is an important factor for cell proliferation, it is not a major factor in the regulation of β‐catenin signaling by PMVK (Figure S2F, Supporting Information).