Furthermore, although Tipe2ΔNK/ΔNK mice displayed superior tumor control over wild‐type mice, MC38 tumor growth was similar between Tbx21ΔNK/ΔNK mice and Tipe2ΔNK/ΔNK; Tbx21ΔNK/ΔNK mice (Figure 4B) and was also similar between EomesΔNK/ΔNK mice and Tipe2ΔNK/ΔNK; EomesΔNK/ΔNK mice (Figure 4C), indicating that the benefits on tumor control from NK‐specific Tipe2 deletion were dependent on NK‐expressed T‐bet and Eomes. Here, EOMES is linked to neoplasm.