Recent genetic studies suggest a protective effect of C-reactive protein (CRP) on AN, which may relate to infection susceptibility given that CRP is not simply a marker of inflammation (Reay et al., 2022; Tylee et al., 2018), as is often characterised, and directly participates in processes like phagocytosis (Dalton et al., 2018a; Del Giudice & Gangestad, 2018). The gene discussed is CRP; the disease is infection.