YTHDF2 and colorectal carcinoma: METTL14‐mediated m6A modification selectively promotes YTHDF2‐dependent processing of pri‐miR‐6769b and pri‐miR‐499a, which facilitates the maturation of corresponding miRNAs that target SLC2A3 and PGAM1, respectively, thereby inhibiting glycolysis in p53‐WT CRC.