In situ hybridization (ISH) and PCR assay showed METTL14 silencing increased pri‐miR‐6769b and pri‐miR‐499a levels, while METTL14 did not modulate the expression of miR‐6769b‐3p/miR‐499a‐3p and pri‐miR‐6769b/pri‐miR‐499a in p53‐MT CRC cells (Figs 4I and EV4G and H). Here, METTL14 is linked to colorectal carcinoma.