PLK1 and non-small cell lung carcinoma: Regardless of the alanine substitution at GSK3β and CK1α (β-cateninmtGSK3β) phosphorylation sites, cells expressing phosphomimetic β-catenin at Ser311 upregulated the levels of CD44, p-PLK1, and mesenchymal factors and promoted cell motility and invasiveness of NSCLC.