For example, in breast tumors, the TME of tumor-bearing mice can induce γδT cells to differentiate into γδT17 cells and produce IL-17 [84]; when the production of IL-17 by Vδ1+ T cells in the lung increases, it promotes the infiltration of inhibitory neutrophils into lung adenocarcinoma and the development of tumors [85, 86]. This evidence concerns the gene IL17A and breast neoplasm.