The loss of RIPK3 or MLKL had no impact on the expansion or attrition of LCMV specific T cells as evidenced by comparable kinetics in virus-specific CD8+ T cells that recognize GP33 and NP396 across both RIPK3 and MLKL-deficient mice compared to WT mice at 8- and 35-days post infection (Fig. 2A, B). The gene discussed is RIPK3; the disease is infection.