Mice bearing SULT2B1b-tumors treated with an anti-CCR2 depleting mAb [34] for five consecutive days were infiltrated by lower numbers of mono-DCs, identified by the expression of Ly6C, CD11b and Sca-1 markers, and failed to control tumor growth (Supplementary Fig. 5A, B). The gene discussed is CCR2; the disease is neoplasm.