Pro‐inflammatory response in hepatic steatosis may be promoted by various stimuli, such as liver enzymes (ALT, AST), oxidative stress, total cholesterol, and triglycerides, and is characterized by increases of TNF‐ɑ and IL‐6 in the liver (Kazankov et al., 2019; Kim et al., 2010). This evidence concerns the gene GPT and fatty liver disease.