It has been reported that exosomal miR‐17‐5p extracted from nasopharyngeal carcinoma cells improves ECs' angiogenic activity via suppressing bone morphogenetic protein (BMP) and activin receptor membrane‐bound inhibitor (BAMBI) expression, which abrogates inhibitory effect on Akt/VEGF‐A signalling, resulting in Akt/VEGF‐A upregulation.71 Here, AKT1 is linked to nasopharyngeal carcinoma.