G3BP1 and viral infectious disease: Surprisingly, in the absence of PABPC1, the formation of G3BP1 foci was abrogated in SeV-infected cells but not after arsenite treatment or poly(I:C) transfection (Fig. 2i and Supplementary Fig. 3G, H), suggesting that PABPC1 has another important function in promoting SG formation exclusively during viral infection.