As we have previously shown that F. tularensis polarises the responding MAIT cell population towards a MAIT-1 phenotype that confers protection from local and systemic infection [12], we next analysed IFN-γ production by WT and Ripk3−/− MAIT cells and non-MAIT αβ T cells during infection in chimeric mice (Fig. S5). Here, RIPK3 is linked to infection.