Inflammatory responses in the lungs were triggered by LPS stimulation and were characterized by the outstanding elevation of IL-6, TNF-α, and IL-1β in the serum, BALF, and lung tissue of ALI mice, and these inflammatory factors played an irreplaceable role in mediating immune responses, which could exacerbate tissue and organ damage, so reducing inflammatory responses was deemed to be an effective strategy for the remedy of ALI [36,37,38]. The gene discussed is TNF; the disease is acute respiratory distress syndrome.