FLII and acquired epidermolysis bullosa: In the end, by targeting retinoid-related orphan receptor α/Akt, Src, P13Kβ, Erk1/2, p38, effector molecules related to neutrophil activation (reactive oxygen species and MMs, as well as the protein Flii) we came closer to better therapeutic results in experimental EBA [5,132,136,137,138,140,141,151].