To explore the extent of this association we additionally quantified SAT-FABP4 expression in a group of non-obese subjects without HTG, since transcription levels of FABP4 have been previously found increased in SAT from subjects with severe obesity [46] and insulin-resistance with respect to insulin-sensitive BMI-matched obese patients [47]. Here, FABP4 is linked to obesity due to melanocortin 4 receptor deficiency.