In both sexes, the percentage of subjects with increased Lp(a) (cut-off of 30 mg/dL) differed depending on the presence of dyslipidemia (40 vs. 51%, p < 0.05 and 35 vs. 47%, p < 0.001 in females and males, respectively) and family history of CAD only in males (41 vs. 49%, p < 0.01) (Table 2). The gene discussed is LPA; the disease is coronary artery disorder.