In the case of endometriosis, NF-κB stimulates the expression of genes that regulate endometriotic cell adhesion, migration, and proliferation, as well as extracellular matrix (ECM) remodeling and inflammation intensity in ectopic endometrium [1,6], whereas in the case of endometrosis, NF-κB seems to take part in the mediation of the expression of genes that stimulate proinflammatory chemokines and inhibit anti-inflammatory cytokine in fibrotic endometrium, as well as stimulating ECM remodeling and chemotaxis [8,9]. Here, NFKB1 is linked to endometriosis.