In the syngeneic YUMMER1.7 model, we confirmed that the PD1 blockade, at a dose of 10 mg/kg, significantly delayed tumor growth in comparison with the isotype control group on day 9 following treatment initiation, potentially induced by the combined effect of an increasing trend in cell death, as assessed using the apoptotic marker caspase-3, and a trend to a decrease in tumor cell proliferation, as shown by the proliferation marker Ki-67, although still with a lack of statistical significance for the sample sizes under study (n = 5–7/group). This evidence concerns the gene PDCD1 and neoplasm.