The relevance of CERS6-AS1 as an oncogene in BCs has been subsequently confirmed in a nude mouse model xenografted with MDA-MB-231 cells overexpressing CERS6-AS1 by Yan et al. [89], who further documented that CERS6-AS1 can promote cell proliferation and inhibit cell apoptosis by downregulating the expression of the tumor suppressor microRNA-125a-5p, which exhibits opposite effects to those of lncRNA CERS6-AS1, and by upregulating the BC susceptibility to gene 1-associated protein 1 (BAP1). This evidence concerns the gene CERS6 and neoplasm.