LCN2 and acute kidney injury: In general, “injury biomarkers” (e.g., NGAL, kidney injury molecule 1 (KIM-1), tissue inhibitor of metalloproteinase-2 (TIMP-2) and insulin-like growth factor binding protein 7 (IGFBP7)) are proposed to be shed by damaged renal structures and, thus, to discriminate AKI forms with variable success [1,44,45].