C1QTNF9 and myocardial infarction: With regard to the heart, administration of globular recombinant CTRP9 (lacking large parts of the N-terminal region) was protective by preventing cardiomyocyte death during ischemia/reperfusion injury in normal and diabetic mice, as well as in remodeling after myocardial infarction (MI) [10,11,12,13].