The knockdown of some molecules involved in TGF-β/EMT signalling could halt this EMT pathway and could be used for cancer treatment; such molecules could be CD36, a membrane glycoprotein present on various epithelial cells [83]; RhoE, a RNA/DNA helicase [84]; FAD104, a fibronectin type III domain-containing protein (FNDC) [87]; p68, a type of RNA helicase [89]; and Sine oculis homeobox homolog 1 (SIX1), a transcription factor associated with development but rarely expressed in adults [91]. This evidence concerns the gene SIX1 and cancer.