MYC and neoplasm: In addition, we showed that upon intranasal administration to a KRASG12D-mutated mouse model of lung adenocarcinoma, the Omomyc miniprotein colocalizes with lung tumors and blocks MYC-driven transcription and proliferation in vivo, where it also induces apoptosis and recruitment of intratumoral immune infiltrates, overall significantly abrogating tumor progression and reducing tumor grading [8].