It has been reported that, in TNBC cells (MDA-MB-231), B7-H6 knockdown can induce apoptosis and inhibit the proliferation of cancer cells, leading to the suppression of tumor development by regulating the T cell-mediated immune response, which indicated that B7-H6 possesses the potential to play a significant role in the treatment of TNBC [108]. This evidence concerns the gene NCR3LG1 and cancer.