In mouse neuroblastoma cell lines, propofol pretreatment ameliorated oxygen-glucose deprivation/reperfusion-induced inflammation and inhibited TNF-α production by modulating the nuclear transcription factor κB (NF-κB) pathway, indicating that propofol may be considered as a potential therapeutic approach to reduce inflammation in neuroblastoma [67]. Here, NFKB1 is linked to neuroblastoma.