One such effort led to the identification of SFN as an anti-proliferative agent against cervical cancer, in which SFN-exposed cervical cancer cells restored the expression of cadherin 1 (CDH1), death-associated protein kinase 1 (DAPK1), retinoic acid receptor (RARβ), and glutathione S-transferase Pi 1 (GSTP1), which was possibly due to the reduction in the expression of HDAC1 and DNMT3b [41]. Here, DNMT3B is linked to cervical carcinoma.