It can not only prevent acetaminophen-induced liver injury by promoting autophagy (Zhang et al., 2020), but also balance the expression of lipid metabolism-related genes by downregulating metabolic disorders and TNF-α/RIPK3 signaling associated with liver inflammation, ultimately achieving the therapeutic effect of inhibiting lipid accumulation and steatohepatitis. This evidence concerns the gene RIPK3 and Other metabolic disease.