To further understand the molecular mechanism by which the FGL2-blocking scFv induces CD69+CD8+ TM cell generation, we analyzed CyTOF data for chemokine receptors (i.e., CCR2, CXCR3, CXCR2, and CX3CR1) that may affect T cell infiltration and recruitment to tumor sites. The gene discussed is CXCR3; the disease is neoplasm.