Asiaticoside‐treated MPTP rat models of Parkinsonism have shown preservation of dopaminergic neurons by inhibiting MPTP‐induced neurotoxicity and also maintaining the metabolic balance of dopamine, protecting against locomotor dysfunction, preventing oxidative damage and upregulation of Bcl‐2 expression while simultaneously reducing levels of pro‐apoptotic protein Bax. The gene discussed is BAX; the disease is Parkinsonism.