(92) similarly used intermittent CGM to initiate and then titrate doses of diazoxide in a patient with Glut1 deficiency who had failed ketogenic diet; without the high granularity data of CGM it was felt that diazoxide would have been administered at incorrect doses, risking the loss of efficacy seen in other cases treated without CGM. This evidence concerns the gene SLC2A1 and hyperinsulinemic hypoglycemia, familial, 4.