Although they used different animal models of NAFLD, Lee et al. investigated FPR2 action in a nonalcoholic steatohepatitis (NASH)-like model induced by CDAHFD feeding for a longer time compared to the HFD treatment period, and Chen’s group examined FPR2’s role in NAFL, a milder disease than NASH. This evidence concerns the gene FPR2 and metabolic dysfunction-associated steatotic liver disease.