The increased type I interferon-stimulated gene expression in BAF-depleted Akata-BX1 cells with reactivating EBV was less dramatic than in the AGS-EBV epithelial cell model, likely due to previously reported dysfunction in cGAS-STING signaling induced by EBV infection in B cells being only partially reversed by BAF depletion67. Here, BANF1 is linked to Epstein-Barr virus infection.