ENO1 and cytomegalovirus infection: (2021) in a murine model of CMV infection, where it was observed that defects in OXPHOS due to cyclooxygenase-2 (COX2) deletion promote the upregulation of glycolytic enzyme genes: LDHA, glyceraldehyde-3-phosphate dehydrogenase (GAPDH), pyruvate kinase muscle isozyme (PKM), enolase 1 (ENO1), phosphoglycerate kinase 1 (PGK1), aldolase A (ALDOA) (69).