ACTG1 and neoplasm: On the contrary, we can observe proteins overexpressed in the tumor compared to tumoroids involved in cell growth and maintenance, actin filament-based process (through ACTG1, ACTN1, ARPC2, EZR, FLNA, GSN, MYH11, MYH14, MYH9, MYL6, MYO1C, TLN1, TPM3, VIM), cell communication, signal transduction, and immune response (Fig. 7E).