Their main clinical features consisted of diverse phenotypes, including meningoencephalomyelitis (n=8), meningoencephalitis (n=3, 1 with diffuse sulcus enhancement), encephalitis (n=4, 1 with diffuse white matter MRI abnormalities, 1 with concurrent EBV DNA), myelitis (n=1), ADEM-like syndrome (n=2), stiff-person spectrum disorder-mimicking syndrome (n=1), encephalopathy (n=1), or overlapping anti-NMDAR encephalitis and MOG-positive demyelinating syndrome (n=1). This evidence concerns the gene MOG and myelitis.