So it was demonstrated that miR-155, abundantly expressed in the lungs of cystic fibrosis patients, indirectly promotes inflammation by driving hyperexpression of CXCL8, but that miR-17 overexpression in cystic fibrosis airway epithelial cells decreases CXCL8 production [71]. Here, CXCL8 is linked to cystic fibrosis.