Since we additionally identified the accessory proteins (DDOST and RPN2) and the catalytic subunits (STT3A and STT3B) of the OST complex as hits, we conclude that N-linked glycosylation plays an important general role in phagocytosis and consequently speculate that dysregulated phagocytosis might be a factor in XMEN disease (Ravell et al, 2020). The gene discussed is STT3B; the disease is X-linked immunodeficiency with magnesium defect, Epstein-Barr virus infection and neoplasia.