Recently, low‐frequency somatic KRAS mutations were detected in human sporadic bAVMs,4, 5, 6, 7, 8 and in vivo experiments have confirmed that KRAS mutation can drive abnormal vascular morphology and arteriovenous malformation (AVM) development in mouse and zebrafish models.9, 10. Here, KRAS is linked to arteriovenous hemangioma/malformation.