Previous studies have reported two main mechanisms of pericytes for promoting tumor growth and metastasis, one is the pericyte‐CAF transition induced by PDGF‐BB‐PDGFRβ signaling,[27] which is also consistent with our observations in the present study that PDGFRB is upregulated in pericytes and similarity between pericytes and CAFs in UMAP clustering plot (Figure 4a,b). The gene discussed is PDGFRB; the disease is neoplasm.