IHC of the in LUAD‐OG1 organoids showed ALK was over‐expressed, and the presence of EML4‐ALK fusion variant 1 was further validated by RT‐PCR and sanger sequencing using specific fusion primers,[25] consistent with the genotype of the parental cancer (Figure 7A–C; Figure S10A, Supporting Information). This evidence concerns the gene ALK and cancer.