We previously reported that nmMLCK participates in intermittent hypoxia‐induced endothelial dysfunction, resulting from IL‐6 secretion and endothelial permeability, in addition to its interaction with the NF‐κB pathway in LPS‐induced vascular inflammation (Recoquillon et al., 2015, 2017). Here, NFKB1 is linked to endothelial dysfunction.